Classical Swine Fever Virus N(pro) Antagonizes IRF3 To Prevent Interferon-Independent TLR3- and RIG-I-Mediated Apoptosis

Lignende værker

• Classical Swine Fever Virus N(pro) Interacts with Interferon Regulatory Factor 3 and Induces Its Proteasomal Degradation
  af: Bauhofer, Oliver, et al.
  Udgivet: (2007)
• Interferon regulatory factor 3-CL, an isoform of IRF3, antagonizes activity of IRF3
  af: Li, Chunhua, et al.
  Udgivet: (2011)
• Classical swine fever virus triggers RIG-I and MDA5-dependent signaling pathway to IRF-3 and NF-κB activation to promote secretion of interferon and inflammatory cytokines in porcine alveolar macrophages
  af: Dong, Xiao-Ying, et al.
  Udgivet: (2013)
• Loss of Interferon Regulatory Factor 3 in Cells Infected with Classical Swine Fever Virus Involves the N-Terminal Protease, N(pro)
  af: La Rocca, S. Anna, et al.
  Udgivet: (2005)
• Classical Swine Fever Virus Can Remain Virulent after Specific Elimination of the Interferon Regulatory Factor 3-Degrading Function of N(pro)
  af: Ruggli, Nicolas, et al.
  Udgivet: (2009)