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Tuning macrophage phenotype to mitigate skeletal muscle fibrosis: Macrophage and FAP Crosstalk in Muscular Polytrauma
Myeloid cells are critical to the development of fibrosis following muscle injury, however, the mechanism of their role in fibrosis formation remains unclear. Here we demonstrate that myeloid cell-derived TGF-β1 signaling is increased in a pro-fibrotic ischemia-reperfusion and cardiotoxin (IR/CTX) m...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | J Immunol |
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| Κύριοι συγγραφείς: | , , , , , , , , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
2020
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8080967/ https://ncbi.nlm.nih.gov/pubmed/32161098 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1900814 |
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