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Quantification of N-terminal amyloid-β isoforms reveals isomers are the most abundant form of the amyloid-β peptide in sporadic Alzheimer’s disease

Plaques that characterize Alzheimer’s disease accumulate over 20 years as a result of decreased clearance of amyloid-β peptides. Such long-lived peptides are subjected to multiple post-translational modifications, in particular isomerization. Using liquid chromatography ion mobility separations mass...

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Podrobná bibliografie
Vydáno v:Brain Commun
Hlavní autoři: Mukherjee, Soumya, Perez, Keyla A, Lago, Larissa C, Klatt, Stephan, McLean, Catriona A, Birchall, Ian E, Barnham, Kevin J, Masters, Colin L, Roberts, Blaine R
Médium: Artigo
Jazyk:Inglês
Vydáno: Oxford University Press 2021
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC8062259/
https://ncbi.nlm.nih.gov/pubmed/33928245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/braincomms/fcab028
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