Cardiomyocyte protective effects of thyroid hormone during hypoxia/reoxygenation injury through activating of IGF‐1‐mediated PI3K/Akt signalling

Ischaemia/reperfusion (I/R) injury is a common clinical condition that results in apoptosis and oxidative stress injury. Thyroid hormone was previously reported to elicit cardiac myocyte hypertrophy and promote cardiac function after cardiac injury. We used an in vivo mouse model of I/R injury and i...

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Veröffentlicht in:J Cell Mol Med
Hauptverfasser: Zeng, Bin, Liu, Lei, Liao, Xiaoting, Zhang, Caixia
Format: Artigo
Sprache:Inglês
Veröffentlicht: John Wiley and Sons Inc. 2021
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Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC8034470/
https://ncbi.nlm.nih.gov/pubmed/33724692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.16389
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