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Potentiation of NETs release is novel characteristic of TREM-1 activation and the pharmacological inhibition of TREM-1 could prevent from the deleterious consequences of NETs release in sepsis

During sepsis, neutrophil activation induces endothelial cell (EC) dysfunction partly through neutrophil extracellular trap (NET) release. The triggering receptor expressed on myeloid cell-1 (TREM-1) is an orphan immune receptor that amplifies the inflammatory response mediated by Toll-like receptor...

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Detalhes bibliográficos
Publicado no:Cell Mol Immunol
Main Authors: Boufenzer, Amir, Carrasco, Kevin, Jolly, Lucie, Brustolin, Benjamin, Di-Pillo, Elisa, Derive, Marc, Gibot, Sébastien
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2021
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC8026640/
https://ncbi.nlm.nih.gov/pubmed/33420354
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41423-020-00591-7
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