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Human Atrial Fibrillation Is Not Associated With Remodeling of Ryanodine Receptor Clusters
The release of Ca(2+) by ryanodine receptor (RyR2) channels is critical for cardiac function. However, abnormal RyR2 activity has been linked to the development of arrhythmias, including increased spontaneous Ca(2+) release in human atrial fibrillation (AF). Clustering properties of RyR2 have been s...
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| I publikationen: | Front Cell Dev Biol |
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| Huvudupphovsmän: | , , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Frontiers Media S.A.
2021
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7947344/ https://ncbi.nlm.nih.gov/pubmed/33718369 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fcell.2021.633704 |
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