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BYSL contributes to tumor growth by cooperating with the mTORC2 complex in gliomas

OBJECTIVE: BYSL, which encodes the Bystin protein in humans, is upregulated in reactive astrocytes following brain damage and/or inflammation. We aimed to determine the role and mechanism of BYSL in glioma cell growth and survival. METHODS: BYSL expression in glioma tissues was measured by quantitat...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Cancer Biol Med
Κύριοι συγγραφείς: Gao, Shangfeng, Sha, Zhuang, Zhou, Junbo, Wu, Yihao, Song, Yunnong, Li, Cheng, Liu, Xuejiao, Zhang, Tong, Yu, Rutong
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: Compuscript 2021
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7877178/
https://ncbi.nlm.nih.gov/pubmed/33628587
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.20892/j.issn.2095-3941.2020.0096
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