RGC-32 Regulates Generation of Reactive Astrocytes in Experimental Autoimmune Encephalomyelitis

Astrocytes are increasingly recognized as critical contributors to multiple sclerosis pathogenesis. We have previously shown that lack of Response Gene to Complement 32 (RGC-32) alters astrocyte morphology in the spinal cord at the peak of experimental autoimmune encephalomyelitis (EAE), suggesting...

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Veröffentlicht in:Front Immunol
Hauptverfasser: Tatomir, Alexandru, Beltrand, Austin, Nguyen, Vinh, Boodhoo, Dallas, Mekala, Armugam, Cudrici, Cornelia, Badea, Tudor C., Muresanu, Dafin F., Rus, Violeta, Rus, Horea
Format: Artigo
Sprache:Inglês
Veröffentlicht: Frontiers Media S.A. 2021
Schlagworte:
Immunology
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7868332/
https://ncbi.nlm.nih.gov/pubmed/33569054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2020.608294
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