RGC-32 Regulates Generation of Reactive Astrocytes in Experimental Autoimmune Encephalomyelitis

Astrocytes are increasingly recognized as critical contributors to multiple sclerosis pathogenesis. We have previously shown that lack of Response Gene to Complement 32 (RGC-32) alters astrocyte morphology in the spinal cord at the peak of experimental autoimmune encephalomyelitis (EAE), suggesting...

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Dettagli Bibliografici
Pubblicato in:Front Immunol
Autori principali: Tatomir, Alexandru, Beltrand, Austin, Nguyen, Vinh, Boodhoo, Dallas, Mekala, Armugam, Cudrici, Cornelia, Badea, Tudor C., Muresanu, Dafin F., Rus, Violeta, Rus, Horea
Natura: Artigo
Lingua:Inglês
Pubblicazione: Frontiers Media S.A. 2021
Soggetti:
Immunology
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7868332/
https://ncbi.nlm.nih.gov/pubmed/33569054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fimmu.2020.608294
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