Global knockout of ROMK potassium channel worsens cardiac ischemia-reperfusion injury but cardiomyocyte-specific knockout does not: implications for the identity of mitoKATP

The renal-outer-medullary-potassium (ROMK) channel, mutated in Bartter’s syndrome, regulates ion exchange in kidney, but its extra-renal functions remain unknown. Additionally, ROMK was postulated to be the pore-forming subunit of the mitochondrial ATP-sensitive K(+) channel (mitoK(ATP)), a mediator...

ver descrição completa

Na minha lista:
Detalhes bibliográficos
Publicado no:J Mol Cell Cardiol
Main Authors: Papanicolaou, Kyriakos N., Ashok, Deepthi, Liu, Ting, Bauer, Tyler M., Sun, Junhui, Li, Zhen, Da Costa, Eduardo, D’Orleans, Charles Crepy, Nathan, Sara, Lefer, David J., Murphy, Elizabeth, Paolocci, Nazareno, Foster, D. Brian, O’Rourke, Brian
Formato: Artigo
Idioma:Inglês
Publicado em: 2020
Assuntos:
Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7849919/
https://ncbi.nlm.nih.gov/pubmed/32004507
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2020.01.010
Tags: Adicionar Tag
Sem tags, seja o primeiro a adicionar uma tag!

Registos relacionados