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Pathogenic mutations in the kinesin-3 motor KIF1A diminish force generation and movement through allosteric mechanisms

The kinesin-3 motor KIF1A functions in neurons, where its fast and superprocessive motility facilitates long-distance transport, but little is known about its force-generating properties. Using optical tweezers, we demonstrate that KIF1A stalls at an opposing load of ~3 pN but more frequently detach...

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Vydáno v:J Cell Biol
Hlavní autoři: Budaitis, Breane G., Jariwala, Shashank, Rao, Lu, Yue, Yang, Sept, David, Verhey, Kristen J., Gennerich, Arne
Médium: Artigo
Jazyk:Inglês
Vydáno: Rockefeller University Press 2021
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC7844421/
https://ncbi.nlm.nih.gov/pubmed/33496723
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.202004227
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