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FSTL1 aggravates cigarette smoke-induced airway inflammation and airway remodeling by regulating autophagy
BACKGROUND: Cigarette smoke (CS) is a major risk factor for Chronic Obstructive Pulmonary Disease (COPD). Follistatin-like protein 1 (FSTL1), a critical factor during embryogenesis particularly in respiratory lung development, is a novel mediator related to inflammation and tissue remodeling. We tri...
Sparad:
| I publikationen: | BMC Pulm Med |
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| Huvudupphovsmän: | , , , , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
BioMed Central
2021
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7841997/ https://ncbi.nlm.nih.gov/pubmed/33509151 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12890-021-01409-6 |
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