Dissemination of Mycobacterium tuberculosis is associated to a SIGLEC1 null variant that limits antigen exchange via trafficking extracellular vesicles

The identification of individuals with null alleles enables studying how the loss of gene function affects infection. We previously described a non‐functional variant in SIGLEC1, which encodes the myeloid‐cell receptor Siglec‐1/CD169 implicated in HIV‐1 cell‐to‐cell transmission. Here we report a si...

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書誌詳細
出版年:J Extracell Vesicles
主要な著者: Benet, Susana, Gálvez, Cristina, Drobniewski, Francis, Kontsevaya, Irina, Arias, Lilibeth, Monguió‐Tortajada, Marta, Erkizia, Itziar, Urrea, Victor, Ong, Ruo‐Yan, Luquin, Marina, Dupont, Maeva, Chojnacki, Jakub, Dalmau, Judith, Cardona, Paula, Neyrolles, Olivier, Lugo‐Villarino, Geanncarlo, Vérollet, Christel, Julián, Esther, Furrer, Hansjakob, Günthard, Huldrych F., Crocker, Paul R., Tapia, Gustavo, Borràs, Francesc E., Fellay, Jacques, McLaren, Paul J., Telenti, Amalio, Cardona, Pere‐Joan, Clotet, Bonaventura, Vilaplana, Cristina, Martinez‐Picado, Javier, Izquierdo‐Useros, Nuria
フォーマット: Artigo
言語:Inglês
出版事項: John Wiley and Sons Inc. 2021
主題:
Research Articles
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC7807485/
https://ncbi.nlm.nih.gov/pubmed/33489013
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/jev2.12046
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