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Neuroprotective effects of ZL006 in Aβ(1–42)-treated neuronal cells

Amyloid beta (Aβ)-induced neurotoxicity and oxidative stress plays an important role in the pathogenesis of Alzheimer’s disease (AD). ZL006 is shown to reduce over-produced nitric oxide and oxidative stress in ischemic stroke by interrupting the interaction of neuronal nitric oxide synthase and post...

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Podrobná bibliografie
Vydáno v:Neural Regen Res
Hlavní autoři: Tao, Wen-Yuan, Yu, Lin-Jie, Jiang, Su, Cao, Xiang, Chen, Jian, Bao, Xin-Yu, Li, Fei, Xu, Yun, Zhu, Xiao-Lei
Médium: Artigo
Jazyk:Inglês
Vydáno: Wolters Kluwer - Medknow 2020
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC7749460/
https://ncbi.nlm.nih.gov/pubmed/32594052
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4103/1673-5374.285006
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