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Neuroprotective effects of ZL006 in Aβ(1–42)-treated neuronal cells
Amyloid beta (Aβ)-induced neurotoxicity and oxidative stress plays an important role in the pathogenesis of Alzheimer’s disease (AD). ZL006 is shown to reduce over-produced nitric oxide and oxidative stress in ischemic stroke by interrupting the interaction of neuronal nitric oxide synthase and post...
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| Publicado no: | Neural Regen Res |
|---|---|
| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Wolters Kluwer - Medknow
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7749460/ https://ncbi.nlm.nih.gov/pubmed/32594052 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4103/1673-5374.285006 |
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