Inhibition of SETMAR–H3K36me2–NHEJ repair axis in residual disease cells prevents glioblastoma recurrence

BACKGROUND: Residual disease of glioblastoma (GBM) causes recurrence. However, targeting residual cells has failed, due to their inaccessibility and our lack of understanding of their survival mechanisms to radiation therapy. Here we deciphered a residual cell–specific survival mechanism essential f...

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Detaylı Bibliyografya
Yayımlandı:Neuro Oncol
Asıl Yazarlar: Kaur, Ekjot, Nair, Jyothi, Ghorai, Atanu, Mishra, Saket V, Achareker, Anagha, Ketkar, Madhura, Sarkar, Debashmita, Salunkhe, Sameer, Rajendra, Jacinth, Gardi, Nilesh, Desai, Sanket, Iyer, Prajish, Thorat, Rahul, Dutt, Amit, Moiyadi, Aliasgar, Dutt, Shilpee
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Oxford University Press 2020
Konular:
Basic and Translational Investigations
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC7746947/
https://ncbi.nlm.nih.gov/pubmed/32458986
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/noaa128
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