Inhibition of SETMAR–H3K36me2–NHEJ repair axis in residual disease cells prevents glioblastoma recurrence

BACKGROUND: Residual disease of glioblastoma (GBM) causes recurrence. However, targeting residual cells has failed, due to their inaccessibility and our lack of understanding of their survival mechanisms to radiation therapy. Here we deciphered a residual cell–specific survival mechanism essential f...

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Bibliografiske detaljer
Udgivet i:Neuro Oncol
Main Authors: Kaur, Ekjot, Nair, Jyothi, Ghorai, Atanu, Mishra, Saket V, Achareker, Anagha, Ketkar, Madhura, Sarkar, Debashmita, Salunkhe, Sameer, Rajendra, Jacinth, Gardi, Nilesh, Desai, Sanket, Iyer, Prajish, Thorat, Rahul, Dutt, Amit, Moiyadi, Aliasgar, Dutt, Shilpee
Format: Artigo
Sprog:Inglês
Udgivet: Oxford University Press 2020
Fag:
Basic and Translational Investigations
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7746947/
https://ncbi.nlm.nih.gov/pubmed/32458986
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/noaa128
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