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ACSS3 represses prostate cancer progression through downregulating lipid droplet-associated protein PLIN3
Current endocrine therapy for prostate cancer (PCa) mainly inhibits androgen/androgen receptor (AR) signaling. However, due to increased intratumoural androgen synthesis and AR variation, PCa progresses to castration-resistant prostate cancer (CRPC), which ultimately becomes resistant to endocrine t...
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| 發表在: | Theranostics |
|---|---|
| Main Authors: | , , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Ivyspring International Publisher
2021
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7738848/ https://ncbi.nlm.nih.gov/pubmed/33391508 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.49384 |
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