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Inhibition of Pyk2 and Src activity improves Cx43 gap junction intercellular communication
Identification of proteins that interact with Cx43 has been instrumental in the understanding of gap junction (GJ) regulation. An in vitro phosphorylation screen identified that Protein tyrosine kinase 2 beta (Pyk2) phosphorylated purified Cx43CT and this led us to characterize the impact of this ph...
Sparad:
| I publikationen: | J Mol Cell Cardiol |
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| Huvudupphovsmän: | , , , , , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2020
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7736531/ https://ncbi.nlm.nih.gov/pubmed/32956670 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2020.09.004 |
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