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Hdac3 deletion in myeloid progenitor cells enhances bone healing in females and limits osteoclast fusion via Pmepa1

Previous studies examining the role of the histone deacetylase Hdac3 within myeloid cells demonstrated that Hdac3 promotes M2 activation and tissue healing in inflammatory conditions. Since myeloid lineage cells are required for proper bone formation and regeneration, in this study we examined the f...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Sci Rep
Egile Nagusiak: Molstad, David H. H., Zars, Elizabeth, Norton, Andrew, Mansky, Kim C., Westendorf, Jennifer J., Bradley, Elizabeth W.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Nature Publishing Group UK 2020
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC7733476/
https://ncbi.nlm.nih.gov/pubmed/33311522
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-020-78364-5
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