Sepsis impedes EAE disease development and diminishes autoantigen-specific naive CD4 T cells

Evaluation of sepsis-induced immunoparalysis has highlighted how decreased lymphocyte number/function contribute to worsened infection/cancer. Yet, an interesting contrast exists with autoimmune disease development, wherein diminishing pathogenic effectors may benefit the post-septic host. Within th...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:eLife
Päätekijät: Jensen, Isaac J, Jensen, Samantha N, Sjaastad, Frances V, Gibson-Corley, Katherine N, Dileepan, Thamothrampillai, Griffith, Thomas S, Mangalam, Ashutosh K, Badovinac, Vladimir P
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: eLife Sciences Publications, Ltd 2020
Aiheet:
Immunology and Inflammation
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC7721438/
https://ncbi.nlm.nih.gov/pubmed/33191915
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7554/eLife.55800
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