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CaMKII binding to GluN2B at S1303 Has No Role in Acute or Inflammatory Pain
Activation of Ca(2+)/calmodulin kinase II (CaMKII) and the N-Methyl D-aspartate receptor (NMDAR), particularly its GluN2B subunit, contribute to the central sensitization of nociceptive pathways and persistent pain. Using mutant mice wherein the activity-driven binding of CaMKII to S1303 in GluN2B i...
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| Publicat a: | Brain Res |
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| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7719074/ https://ncbi.nlm.nih.gov/pubmed/33068634 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2020.147154 |
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