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Mitochondrial-Protective Effects of R-Phenibut after Experimental Traumatic Brain Injury
Altered neuronal Ca(2+) homeostasis and mitochondrial dysfunction play a central role in the pathogenesis of traumatic brain injury (TBI). R-Phenibut ((3R)-phenyl-4-aminobutyric acid) is an antagonist of the α(2)δ subunit of voltage-dependent calcium channels (VDCC) and an agonist of gamma-aminobuty...
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| Izdano u: | Oxid Med Cell Longev |
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| Glavni autori: | , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Hindawi
2020
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7700030/ https://ncbi.nlm.nih.gov/pubmed/33274011 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2020/9364598 |
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