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YTHDF2 mediates the mRNA degradation of the tumor suppressors to induce AKT phosphorylation in N6-methyladenosine-dependent way in prostate cancer
BACKGROUND: N6-methyladenosine (m(6)A) is the most abundant modification in mRNA of humans. Emerging evidence has supported the fact that m(6)A is comprehensively involved in various diseases especially cancers. As a crucial reader, YTHDF2 usually mediates the degradation of m(6)A-modified mRNAs in...
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| 出版年: | Mol Cancer |
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| 主要な著者: | , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7599101/ https://ncbi.nlm.nih.gov/pubmed/33121495 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12943-020-01267-6 |
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