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G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer’s disease pathology
Receptor for advanced glycation end products (RAGE) has been implicated in the pathophysiology of Alzheimers disease(AD) due to its ability to bind amyloid-beta (Aβ42) and mediate inflammatory response. G82S RAGE polymorphism is associated with AD but the molecular mechanism for this association is...
Guardat en:
| Publicat a: | PLoS One |
|---|---|
| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Public Library of Science
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7595441/ https://ncbi.nlm.nih.gov/pubmed/33119615 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0225487 |
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