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G82S RAGE polymorphism influences amyloid-RAGE interactions relevant in Alzheimer’s disease pathology
Receptor for advanced glycation end products (RAGE) has been implicated in the pathophysiology of Alzheimers disease(AD) due to its ability to bind amyloid-beta (Aβ42) and mediate inflammatory response. G82S RAGE polymorphism is associated with AD but the molecular mechanism for this association is...
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| Publicado no: | PLoS One |
|---|---|
| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Public Library of Science
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7595441/ https://ncbi.nlm.nih.gov/pubmed/33119615 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0225487 |
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