Cardiomyocyte-derived small extracellular vesicles can signal eNOS activation in cardiac microvascular endothelial cells to protect against Ischemia/Reperfusion injury

Rationale: The crosstalk between cardiac microvascular endothelial cells (CMECs) and cardiomyocytes (CMs) has emerged as a key component in the development of, and protection against, cardiac diseases. For example, activation of endothelial nitric oxide synthase (eNOS) in CMECs, by therapeutic strat...

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Dades bibliogràfiques
Publicat a:Theranostics
Autors principals: Chen, Guihao, Xu, Chuansheng, Gillette, Thomas G., Huang, Tongyi, Huang, Peisen, Li, Qing, Li, Xiangdong, Li, Qinfeng, Ning, Yu, Tang, Ruijie, Huang, Cunrong, Xiong, Yuyan, Tian, Xiaqiu, Xu, Jun, Xu, Junyan, Chang, Liping, Wei, Cong, Jin, Chen, Hill, Joseph A., Yang, Yuejin
Format: Artigo
Idioma:Inglês
Publicat: Ivyspring International Publisher 2020
Matèries:
Research Paper
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7546010/
https://ncbi.nlm.nih.gov/pubmed/33052245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.43163
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