Cardiomyocyte-derived small extracellular vesicles can signal eNOS activation in cardiac microvascular endothelial cells to protect against Ischemia/Reperfusion injury

Rationale: The crosstalk between cardiac microvascular endothelial cells (CMECs) and cardiomyocytes (CMs) has emerged as a key component in the development of, and protection against, cardiac diseases. For example, activation of endothelial nitric oxide synthase (eNOS) in CMECs, by therapeutic strat...

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Detaylı Bibliyografya
Yayımlandı:Theranostics
Asıl Yazarlar: Chen, Guihao, Xu, Chuansheng, Gillette, Thomas G., Huang, Tongyi, Huang, Peisen, Li, Qing, Li, Xiangdong, Li, Qinfeng, Ning, Yu, Tang, Ruijie, Huang, Cunrong, Xiong, Yuyan, Tian, Xiaqiu, Xu, Jun, Xu, Junyan, Chang, Liping, Wei, Cong, Jin, Chen, Hill, Joseph A., Yang, Yuejin
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Ivyspring International Publisher 2020
Konular:
Research Paper
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC7546010/
https://ncbi.nlm.nih.gov/pubmed/33052245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.7150/thno.43163
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