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Interference with pancreatic sympathetic signaling halts the onset of diabetes in mice
The notably lobular distribution of immune lesions in type 1 diabetes (T1D) has been hypothesized to be the result of innervation within the pancreas. To investigate whether neuroimmune interactions could explain this phenomenon, we explored the impact of sympathetic signaling in the RIP-LCMV-GP mou...
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| Publicado en: | Sci Adv |
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| Autores principales: | , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Association for the Advancement of Science
2020
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7531904/ https://ncbi.nlm.nih.gov/pubmed/33052874 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/sciadv.abb2878 |
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