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Slow train coming: an anti-CCN2 strategy reverses a model of chronic overuse muscle fibrosis
One of the first targets proposed as an anti-fibrotic therapy was CCN2. Proof of its involvement in fibrosis was initially difficult, due to the lack of appropriate reagents and general understanding of the molecular mechanisms responsible for persistent fibrosis. As these issues have been progressi...
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| 發表在: | J Cell Commun Signal |
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| 主要作者: | |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Springer Netherlands
2020
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7511481/ https://ncbi.nlm.nih.gov/pubmed/32410169 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12079-020-00568-1 |
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