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Airborne PAHs inhibit gap junctional intercellular communication and activate MAPKs in human bronchial epithelial cell line
Inhalation exposures to polycyclic aromatic hydrocarbons (PAHs) have been associated with various adverse health effects, including chronic lung diseases and cancer. Using human bronchial epithelial cell line HBE1, we investigated the effects of structurally different PAHs on tissue homeostatic proc...
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| 出版年: | Environ Toxicol Pharmacol |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7486243/ https://ncbi.nlm.nih.gov/pubmed/32492535 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.etap.2020.103422 |
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