טוען...
Molecular changes evoked by the beta-lactam antibiotic ceftriaxone across rodent models of substance use disorder and neurological disease
Ceftriaxone is a beta-lactam antibiotic that increases the expression of the major glutamate transporter, GLT-1. As such, ceftriaxone ameliorates symptoms across multiple rodent models of neurological diseases and substance use disorders. However, the mechanism behind GLT-1 upregulation is unknown....
שמור ב:
| הוצא לאור ב: | Neurosci Biobehav Rev |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2020
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7483567/ https://ncbi.nlm.nih.gov/pubmed/32485268 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neubiorev.2020.05.016 |
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