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Mitochondrial CaMKII causes adverse metabolic reprogramming and dilated cardiomyopathy
Despite the clear association between myocardial injury, heart failure and depressed myocardial energetics, little is known about upstream signals responsible for remodeling myocardial metabolism after pathological stress. Here, we report increased mitochondrial calmodulin kinase II (CaMKII) activat...
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| Publicado no: | Nat Commun |
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| Main Authors: | , , , , , , , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7473864/ https://ncbi.nlm.nih.gov/pubmed/32887881 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-020-18165-6 |
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