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Mitochondrial CaMKII causes adverse metabolic reprogramming and dilated cardiomyopathy

Despite the clear association between myocardial injury, heart failure and depressed myocardial energetics, little is known about upstream signals responsible for remodeling myocardial metabolism after pathological stress. Here, we report increased mitochondrial calmodulin kinase II (CaMKII) activat...

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Detalhes bibliográficos
Publicado no:Nat Commun
Main Authors: Luczak, Elizabeth D., Wu, Yuejin, Granger, Jonathan M., Joiner, Mei-ling A., Wilson, Nicholas R., Gupta, Ashish, Umapathi, Priya, Murphy, Kevin R., Reyes Gaido, Oscar E., Sabet, Amin, Corradini, Eleonora, Tseng, Wen-Wei, Wang, Yibin, Heck, Albert J. R., Wei, An-Chi, Weiss, Robert G., Anderson, Mark E.
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2020
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7473864/
https://ncbi.nlm.nih.gov/pubmed/32887881
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-020-18165-6
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