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Loss of TET2 affects proliferation and drug sensitivity through altered dynamics of cell-state transitions
A persistent puzzle in cancer biology is how mutations, which neither alter growth signaling pathways nor directly interfere with drug mechanism, can still recur and persist in tumors. One example is the mutation of the DNA demethylase TET2 in acute myeloid leukemias (AMLs) that frequently persists...
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| Udgivet i: | Cell Syst |
|---|---|
| Main Authors: | , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
2020
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7428059/ https://ncbi.nlm.nih.gov/pubmed/32619551 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cels.2020.06.003 |
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