N-acetyl cysteine treatment preserves mitochondrial indices of functionality in the brain of hyperammonemic mice

AIM OF THE STUDY: Acute or chronic live failure could result in hyperammonemia and hepatic encephalopathy (HE). HE is a clinical complication characterized by severe cognitive dysfunction and coma. The ammonium ion (NH(4)(+)) is the most suspected toxic molecule involved in the pathogenesis of HE. N...

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Dades bibliogràfiques
Publicat a:Clin Exp Hepatol
Autors principals: Mohammadi, Hamidreza, Sayad, Abolfazl, Mohammadi, Mohsen, Niknahad, Hossein, Heidari, Reza
Format: Artigo
Idioma:Inglês
Publicat: Termedia Publishing House 2020
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Original Paper
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC7380475/
https://ncbi.nlm.nih.gov/pubmed/32728627
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.5114/ceh.2020.95814
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