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N-acetyl cysteine treatment preserves mitochondrial indices of functionality in the brain of hyperammonemic mice
AIM OF THE STUDY: Acute or chronic live failure could result in hyperammonemia and hepatic encephalopathy (HE). HE is a clinical complication characterized by severe cognitive dysfunction and coma. The ammonium ion (NH(4)(+)) is the most suspected toxic molecule involved in the pathogenesis of HE. N...
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| 出版年: | Clin Exp Hepatol |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Termedia Publishing House
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7380475/ https://ncbi.nlm.nih.gov/pubmed/32728627 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.5114/ceh.2020.95814 |
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