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Nicotinic acetylcholine receptor signaling regulates inositol‐requiring enzyme 1α activation to protect β‐cells against terminal unfolded protein response under irremediable endoplasmic reticulum stress

AIMS/INTRODUCTION: Under irremediable endoplasmic reticulum (ER) stress, hyperactivated inositol‐requiring enzyme 1α (IRE1α) triggers the terminal unfolded protein response (T‐UPR), causing crucial cell dysfunction and apoptosis. We hypothesized that nicotinic acetylcholine receptor (nAChR) signalin...

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Bibliografiske detaljer
Udgivet i:J Diabetes Investig
Main Authors: Ishibashi, Tatsuya, Morita, Shuhei, Kishimoto, Shohei, Uraki, Shinsuke, Takeshima, Ken, Furukawa, Yasushi, Inaba, Hidefumi, Ariyasu, Hiroyuki, Iwakura, Hiroshi, Furuta, Hiroto, Nishi, Masahiro, Papa, Feroz R, Akamizu, Takashi
Format: Artigo
Sprog:Inglês
Udgivet: John Wiley and Sons Inc. 2020
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7378412/
https://ncbi.nlm.nih.gov/pubmed/31925927
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jdi.13211
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