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Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia
Niche hijack by malignant cells is considered to be a prominent cause of disease relapse. Barbier and colleagues uncover (E)-selectin as a novel mediator of malignant cell survival and regeneration which, upon blockade, has the potential to significantly improve therapeutic outcomes.
Guardado en:
| Publicado en: | Nat Commun |
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| Autores principales: | , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
Nature Publishing Group UK
2020
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7378234/ https://ncbi.nlm.nih.gov/pubmed/32703951 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-020-17594-7 |
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