The diabetic microenvironment causes mitochondrial oxidative stress in glomerular endothelial cells and pathological crosstalk with podocytes

BACKGROUND: In the setting of diabetes mellitus, mitochondrial dysfunction and oxidative stress are important pathogenic mechanisms causing end organ damage, including diabetic kidney disease (DKD), but mechanistic understanding at a cellular level remains obscure. In mouse models of DKD, glomerular...

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Bibliografiska uppgifter
I publikationen:Cell Commun Signal
Huvudupphovsmän: Casalena, Gabriella A., Yu, Liping, Gil, Roberto, Rodriguez, Samuel, Sosa, Shantel, Janssen, William, Azeloglu, Evren U., Leventhal, Jeremy S., Daehn, Ilse S.
Materialtyp: Artigo
Språk:Inglês
Publicerad: BioMed Central 2020
Ämnen:
Research
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC7341607/
https://ncbi.nlm.nih.gov/pubmed/32641054
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12964-020-00605-x
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