Behavioral deficits and cellular damage following developmental ethanol exposure in rats are attenuated by CP-101,606, an NMDAR antagonist with unique NR2B specificity

NMDAR-mediated excitotoxicity has been implicated in some of the impairments following fetal ethanol exposure. Previous studies suggest that both neuronal cell death and some of the behavioral deficits can be reduced by NMDAR antagonism during withdrawal, including antagonism of a subpopulation of r...

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Veröffentlicht in:Pharmacol Biochem Behav
Hauptverfasser: Lewis, B., Wellmann, K.A., Kehrberg, A.M.H., Carter, M.L., Baldwin, T., Cohen, M., Barron, S.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2011
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Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7307382/
https://ncbi.nlm.nih.gov/pubmed/22037411
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.pbb.2011.10.013
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