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Nicotinamide mononucleotide attenuates glucocorticoid-induced osteogenic inhibition by regulating the SIRT1/PGC-1α signaling pathway
Long-term and high-dose glucocorticoid treatment is recognized as an important influencing factor for osteoporosis and osteonecrosis. Nicotinamide mononucleotide (NMN) is an intermediate of NAD(+) biosynthesis, and is widely used to replenish the levels of NAD(+). However, the potential role of NMN...
Guardat en:
| Publicat a: | Mol Med Rep |
|---|---|
| Autors principals: | , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
D.A. Spandidos
2020
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7248519/ https://ncbi.nlm.nih.gov/pubmed/32377728 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/mmr.2020.11116 |
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