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NF-κB mediates lipopolysaccharide-induced alternative pre-mRNA splicing of MyD88 in mouse macrophages

Although a robust inflammatory response is needed to combat infection, this response must ultimately be terminated to prevent chronic inflammation. One mechanism that terminates inflammatory signaling is the production of alternative mRNA splice forms in the Toll-like receptor (TLR) signaling pathwa...

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Bibliographische Detailangaben
Veröffentlicht in:J Biol Chem
Hauptverfasser: Lee, Frank Fang-Yao, Davidson, Kevin, Harris, Chelsea, McClendon, Jazalle, Janssen, William J., Alper, Scott
Format: Artigo
Sprache:Inglês
Veröffentlicht: American Society for Biochemistry and Molecular Biology 2020
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC7196638/
https://ncbi.nlm.nih.gov/pubmed/32179652
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA119.011495
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