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NF-κB mediates lipopolysaccharide-induced alternative pre-mRNA splicing of MyD88 in mouse macrophages

Although a robust inflammatory response is needed to combat infection, this response must ultimately be terminated to prevent chronic inflammation. One mechanism that terminates inflammatory signaling is the production of alternative mRNA splice forms in the Toll-like receptor (TLR) signaling pathwa...

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Dettagli Bibliografici
Pubblicato in:J Biol Chem
Autori principali: Lee, Frank Fang-Yao, Davidson, Kevin, Harris, Chelsea, McClendon, Jazalle, Janssen, William J., Alper, Scott
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Biochemistry and Molecular Biology 2020
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC7196638/
https://ncbi.nlm.nih.gov/pubmed/32179652
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA119.011495
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