Staphylococcus aureus colonization is increased on lupus skin lesions and is promoted by interferon-mediated barrier disruption

Cutaneous inflammation is recurrent in systemic lupus erythematosus (SLE), yet mechanisms that drive cutaneous inflammation in SLE are not well-defined. Type I IFNs are elevated in non-lesional SLE skin and promote inflammatory responses. Staphylococcus aureus, known to induce IFN production, could...

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Опубликовано в: :J Invest Dermatol
Главные авторы: Sirobhushanam, Sirisha, Parsa, Navya, Reed, Tamra J., Berthier, Celine C., Sarkar, Mrinal K., Hile, Grace A., Tsoi, Lam C., Banfield, Josh, Dobry, Craig, Horswill, Alexander R., Gudjonsson, Johann E., Kahlenberg, J. Michelle
Формат: Artigo
Язык:Inglês
Опубликовано: 2019
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Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC7183889/
https://ncbi.nlm.nih.gov/pubmed/31877319
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.jid.2019.11.016
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