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PI3K activation prevents Aβ42-induced synapse loss and favors insoluble amyloid deposit formation
Excess of Aβ42 peptide is considered a hallmark of the disease. Here we express the human Aβ42 peptide to assay the neuroprotective effects of PI3K in adult Drosophila melanogaster. The neuronal expression of the human peptide elicits progressive toxicity in the adult fly. The pathological traits in...
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| Publicado no: | Mol Biol Cell |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The American Society for Cell Biology
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7183762/ https://ncbi.nlm.nih.gov/pubmed/31877058 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E19-05-0303 |
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