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CCN1 triggers adaptive autophagy in cardiomyocytes to curb its apoptotic activities
Autophagy occurs at basal levels for cellular homeostasis under normal conditions and is increased in response to nutrient starvation or stress to ensure cell survival. However, excessive autophagy can be deleterious to cardiomyocytes. CCN1/Cyr61, a matricellular protein, is expressed in the stresse...
שמור ב:
| הוצא לאור ב: | J Cell Commun Signal |
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| Main Authors: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Springer Netherlands
2019
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7176774/ https://ncbi.nlm.nih.gov/pubmed/31659628 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s12079-019-00534-6 |
| תגים: |
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