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Genetic Suppression of mTOR Rescues Synaptic and Social Behavioral Abnormalities in a Mouse Model of Pten Haploinsufficiency

Heterozygous mutations in PTEN, which encodes a negative regulator of the mTOR and β-catenin signaling pathways, cause macrocephaly/autism syndrome. However, the neurobiological substrates of the core symptoms of this syndrome are poorly understood. Here, we investigate the relationship between cere...

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Publicado en:Autism Res
Main Authors: Huang, Wen-Chin, Chen, Youjun, Page, Damon T.
Formato: Artigo
Idioma:Inglês
Publicado: 2019
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC7141489/
https://ncbi.nlm.nih.gov/pubmed/31441226
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/aur.2186
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