Downregulation of Paralemmin-3 Ameliorates Lipopolysaccharide-Induced Acute Lung Injury in Rats by Regulating Inflammatory Response and Inhibiting Formation of TLR4/MyD88 and TLR4/TRIF Complexes

Previous studies have demonstrated paralemmin-3 (PALM3) participates in Toll-like receptor (TLR) signaling. This study investigated the effect of PALM3 knockdown on lipopolysaccharide (LPS)-induced acute lung injury (ALI) and its underlying mechanisms. We constructed a recombinant adenoviral vector...

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Detalhes bibliográficos
Publicado no:Inflammation
Main Authors: Chen, Xuxin, Tang, Lu, Feng, Jian, Wang, Yi, Han, Zhihai, Meng, Jiguang
Formato: Artigo
Idioma:Inglês
Publicado em: Springer US 2017
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Original Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC7102376/
https://ncbi.nlm.nih.gov/pubmed/28801798
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10753-017-0639-9
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