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Sepsis Erodes CD8(+) Memory T Cell Protective Immunity Against an EBV Homolog in a 2B4-Dependent Manner
Epstein–Barr virus (EBV) reactivation commonly occurs following sepsis but the mechanisms underlying this are unknown. We utilized a murine EBV homolog (gHV) and the cecal ligation and puncture model of polymicrobial sepsis to study the impact of sepsis on gHV reactivation and CD8(+) T cell immune s...
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| Vydáno v: | J Leukoc Biol |
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| Hlavní autoři: | , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7094871/ https://ncbi.nlm.nih.gov/pubmed/30624806 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/JLB.4A0718-292R |
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