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Augmenting Vacuolar H(+)-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction
The diabetic heart is characterized by a shift in substrate utilization from glucose to lipids, which may ultimately lead to contractile dysfunction. This substrate shift is facilitated by increased translocation of lipid transporter CD36 (SR-B2) from endosomes to the sarcolemma resulting in increas...
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| Vydáno v: | Int J Mol Sci |
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| Hlavní autoři: | , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
MDPI
2020
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7073192/ https://ncbi.nlm.nih.gov/pubmed/32102213 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms21041520 |
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