Augmenting Vacuolar H(+)-ATPase Function Prevents Cardiomyocytes from Lipid-Overload Induced Dysfunction

The diabetic heart is characterized by a shift in substrate utilization from glucose to lipids, which may ultimately lead to contractile dysfunction. This substrate shift is facilitated by increased translocation of lipid transporter CD36 (SR-B2) from endosomes to the sarcolemma resulting in increas...

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Bibliographic Details
Published in:Int J Mol Sci
Main Authors: Wang, Shujin, Wong, Li-Yen, Neumann, Dietbert, Liu, Yilin, Sun, Aomin, Antoons, Gudrun, Strzelecka, Agnieszka, Glatz, Jan F.C., Nabben, Miranda, Luiken, Joost J.F.P.
Format: Artigo
Language:Inglês
Published: MDPI 2020
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC7073192/
https://ncbi.nlm.nih.gov/pubmed/32102213
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/ijms21041520
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