Knocking-out the Siah2 E3 ubiquitin ligase prevents mitochondrial NCX3 degradation, regulates mitochondrial fission and fusion, and restores mitochondrial function in hypoxic neurons

Lignende værker

• Endoplasmic reticulum refilling and mitochondrial calcium extrusion promoted in neurons by NCX1 and NCX3 in ischemic preconditioning are determinant for neuroprotection
  af: Sisalli, M J, et al.
  Udgivet: (2014)
• NCX1 and NCX3 as potential factors contributing to neurodegeneration and neuroinflammation in the A53T transgenic mouse model of Parkinson’s Disease
  af: Sirabella, Rossana, et al.
  Udgivet: (2018)
• A New Cell-penetrating Peptide That Blocks the Autoinhibitory XIP Domain of NCX1 and Enhances Antiporter Activity
  af: Molinaro, Pasquale, et al.
  Udgivet: (2015)
• The NCX3 isoform of the Na(+)/Ca(2+) exchanger contributes to neuroprotection elicited by ischemic postconditioning
  af: Pignataro, Giuseppe, et al.
  Udgivet: (2011)
• Na(+)–Ca(2+) Exchanger (NCX3) Knock-Out Mice Display an Impairment in Hippocampal Long-Term Potentiation and Spatial Learning and Memory
  af: Molinaro, Pasquale, et al.
  Udgivet: (2011)